Haemoglobin E disease (Q39522): Difference between revisions

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Doença da hemoglobina E é caracterizada pela síntese de uma hemoglobina anormal chamada hemoglobina E (Hb E), em vez da hemoglobina A normal (Hb A). Indivíduos heterozigotos para Hb E (AE) têm uma condição assintomática sem relevância clínica, exceto pelo risco de transmitir talassemia E/beta talassemia se o outro progenitor for portador da beta talassemia. A gravidade destas formas de E/beta talassemia é muito variável, e o quadro clínico varia daquela do traço da talassemia minor até talassemia intermédia e talassemia maior. Os indivíduos homozigotos para Hb E (EE) são assintomáticos.
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Haemoglobin E disease is characterised by the synthesis of an abnormal haemoglobin called haemoglobin E (HbE), instead of the normal haemoglobin A (HbA). Subjects heterozygous for HbE (AE) have an asymptomatic condition with no clinical relevance, except for the risk of transmitting E/beta thalassemia if the other parent carries beta thalassemia. The severity of these E/beta thalassemia forms is very variable, the clinical picture ranging from that of beta thalassemia minor through to thalassemia intermedia to thalassemia major. Subjects homozygous for HbE (EE) are asymptomatic.

Revision as of 04:58, 13 August 2026

Haemoglobin E disease is characterised by the synthesis of an abnormal haemoglobin called haemoglobin E (HbE), instead of the normal haemoglobin A (HbA). Subjects heterozygous for HbE (AE) have an asymptomatic condition with no clinical relevance, except for the risk of transmitting E/beta thalassemia if the other parent carries beta thalassemia. The severity of these E/beta thalassemia forms is very variable, the clinical picture ranging from that of beta thalassemia minor through to thalassemia intermedia to thalassemia major. Subjects homozygous for HbE (EE) are asymptomatic.
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3A51.A
    English
    Haemoglobin E disease
    Haemoglobin E disease is characterised by the synthesis of an abnormal haemoglobin called haemoglobin E (HbE), instead of the normal haemoglobin A (HbA). Subjects heterozygous for HbE (AE) have an asymptomatic condition with no clinical relevance, except for the risk of transmitting E/beta thalassemia if the other parent carries beta thalassemia. The severity of these E/beta thalassemia forms is very variable, the clinical picture ranging from that of beta thalassemia minor through to thalassemia intermedia to thalassemia major. Subjects homozygous for HbE (EE) are asymptomatic.

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