Diffuse actinic keratinocyte dysplasia (Q43976): Difference between revisions

From determinar.ia.br - Determine suas informações
‎Changed label, description and/or aliases in pt-br, en
‎Changed an Item
 
(6 intermediate revisions by the same user not shown)
Property / Canonical URI
 
Property / Canonical URI: https://id.who.int/icd/entity/1976997748 / rank
 
Normal rank
Property / CURIE
 
CID11:EK90.1
Property / CURIE: CID11:EK90.1 / rank
 
Normal rank
Property / Canary Token
 
dki-india-EK90.1
Property / Canary Token: dki-india-EK90.1 / rank
 
Normal rank
Property / Verification Status
 
Concluído
Property / Verification Status: Concluído / rank
 
Normal rank
Property / Knowledge Architect
 
Property / Knowledge Architect: https://pauloleads.com.br/cases-publicos/ / rank
 
Normal rank
Property / Collection date
 
13 August 2026
Timestamp+2026-08-13T00:00:00Z
Timezone+00:00
CalendarGregorian
Precision1 day
Before0
After0
Property / Collection date: 13 August 2026 / rank
 
Normal rank
Property / Linked ICD 10
 
Property / Linked ICD 10: L57.0 / rank
 
Normal rank

Latest revision as of 11:13, 13 August 2026

Diffuse actinic dysplasia develops after repeated exposure of skin to ultraviolet radiation, usually over decades, and results from cumulative DNA damage within the nuclei of epidermal keratinocytes. It is characterised initially by subtle diffuse skin changes including mottling, erythema, telangiectasia and irregular fine scaling. It is seen most commonly on the unprotected scalp skin of bald men. Histologically there are early signs of dysplasia in the basal epidermis. As damage accumulates the clinical changes become more pronounced with the formation of discrete actinic keratoses, from which intraepidermal or invasive squamous cell carcinoma may develop.
Language Label Description Also known as
default for all languages
EK90.1
    English
    Diffuse actinic keratinocyte dysplasia
    Diffuse actinic dysplasia develops after repeated exposure of skin to ultraviolet radiation, usually over decades, and results from cumulative DNA damage within the nuclei of epidermal keratinocytes. It is characterised initially by subtle diffuse skin changes including mottling, erythema, telangiectasia and irregular fine scaling. It is seen most commonly on the unprotected scalp skin of bald men. Histologically there are early signs of dysplasia in the basal epidermis. As damage accumulates the clinical changes become more pronounced with the formation of discrete actinic keratoses, from which intraepidermal or invasive squamous cell carcinoma may develop.

      Statements

      CID11:EK90.1
      0 references
      dki-india-EK90.1
      0 references
      Concluído
      0 references
      13 August 2026
      0 references
      0 references