Glomerular disorders in organ-limited amyloidosis (Q104611): Difference between revisions
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Refere-se a transtornos glomerulares em uma categoria de amiloidose na qual a deposição de amiloide clinicamente é limitada a um órgão. É comparada à amiloidose sistêmica e pode ser causada por vários tipos diferentes de amiloide. Em quase todas as patologias específicas de órgãos, há um debate sobre se as placas amilóides são o agente causal da doença ou, consequência a jusante de um agente idiopático comum. | |||
| description / en | description / en | ||
This refers to glomerular disorders in a category of amyloidosis where the distribution can be associated primarily with a single organ. It is contrasted to systemic amyloidosis, and it can be caused by several different types of amyloid. In almost all of the organ-specific pathologies, there is significant debate as to whether the amyloid plaques are the causal agent of the disease or instead a downstream consequence of a common idiopathic agent. | |||
Revision as of 10:28, 17 August 2026
This refers to glomerular disorders in a category of amyloidosis where the distribution can be associated primarily with a single organ. It is contrasted to systemic amyloidosis, and it can be caused by several different types of amyloid. In almost all of the organ-specific pathologies, there is significant debate as to whether the amyloid plaques are the causal agent of the disease or instead a downstream consequence of a common idiopathic agent.
| Language | Label | Description | Also known as |
|---|---|---|---|
| default for all languages | ID_1536765035 |
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| English | Glomerular disorders in organ-limited amyloidosis |
This refers to glomerular disorders in a category of amyloidosis where the distribution can be associated primarily with a single organ. It is contrasted to systemic amyloidosis, and it can be caused by several different types of amyloid. In almost all of the organ-specific pathologies, there is significant debate as to whether the amyloid plaques are the causal agent of the disease or instead a downstream consequence of a common idiopathic agent. |
