Non-amnestic Alzheimer disease dementia, visuospatial variant (Q96047): Difference between revisions

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Variante clínica da demência da doença de Alzheimer, que se apresenta com déficits predominantes de percepção visual e/ou visuoespaciais com função visual primária intacta. Manifestações clínicas incluem elementos da síndrome de Balint (ataxia óptica, apraxia ocular e simultagnosia) associados ou não à síndrome de Gerstmann (acalculia, agrafia, desorientação direita-esquerda e agnosia digital). A memória episódica está proporcionalmente menos comprometida. Há atrofia proeminente nas regiões corticais posteriores à ressonância magnética e hipometabolismo no exame FDG-PET.
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Clinical variant of Alzheimer disease dementia presenting with predominant visual perception and/or visuospatial deficits with intact primary visual function. Clinical features include elements of Balint’s syndrome (optic ataxia, ocular apraxia and simultagnosia) associated or not with Gerstmann’s syndrome (acalculia, agraphia, left–right disorientation and finger agnosia) Episodic memory is proportionally less impaired. There is prominent atrophy in the posterior cortical regions on magnetic resonance and hypometabolism on FDG-PET examination.

Revision as of 11:53, 16 August 2026

Clinical variant of Alzheimer disease dementia presenting with predominant visual perception and/or visuospatial deficits with intact primary visual function. Clinical features include elements of Balint’s syndrome (optic ataxia, ocular apraxia and simultagnosia) associated or not with Gerstmann’s syndrome (acalculia, agraphia, left–right disorientation and finger agnosia) Episodic memory is proportionally less impaired. There is prominent atrophy in the posterior cortical regions on magnetic resonance and hypometabolism on FDG-PET examination.
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    Non-amnestic Alzheimer disease dementia, visuospatial variant
    Clinical variant of Alzheimer disease dementia presenting with predominant visual perception and/or visuospatial deficits with intact primary visual function. Clinical features include elements of Balint’s syndrome (optic ataxia, ocular apraxia and simultagnosia) associated or not with Gerstmann’s syndrome (acalculia, agraphia, left–right disorientation and finger agnosia) Episodic memory is proportionally less impaired. There is prominent atrophy in the posterior cortical regions on magnetic resonance and hypometabolism on FDG-PET examination.

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