Chronic alcoholic myopathy (Q93213): Difference between revisions

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A miopatia alcoólica crônica é uma complicação comum do alcoolismo que afeta aproximadamente 50% dos pacientes. Afeta principalmente as fibras musculares brancas das cinturas peitoral e pélvica, embora outros grupos musculares possam estar envolvidos. Aproximadamente 50% dos pacientes afetados apresentam fraqueza muscular proximal. Não está associado a deficiências nutricionais, de vitaminas ou minerais. A atrofia das fibras musculares não leva a um aumento da creatinoquinase sérica e é reversível com 6-12 meses de abstinência.
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Chronic alcoholic myopathy is a common complication of alcoholism affecting approximately 50% of patients. It primarily affects the white muscle fibres of the pectoral and pelvic girdles, though other muscle groups may be involved. Approximately 50% of affected patients show proximal muscle weakness. It is not associated with nutritional, vitamin or mineral deficiencies. The atrophy of the muscle fibres does not lead to an increase in serum creatine kinase and is reversible with 6-12 months abstinence.

Revision as of 08:19, 16 August 2026

Chronic alcoholic myopathy is a common complication of alcoholism affecting approximately 50% of patients. It primarily affects the white muscle fibres of the pectoral and pelvic girdles, though other muscle groups may be involved. Approximately 50% of affected patients show proximal muscle weakness. It is not associated with nutritional, vitamin or mineral deficiencies. The atrophy of the muscle fibres does not lead to an increase in serum creatine kinase and is reversible with 6-12 months abstinence.
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    Chronic alcoholic myopathy
    Chronic alcoholic myopathy is a common complication of alcoholism affecting approximately 50% of patients. It primarily affects the white muscle fibres of the pectoral and pelvic girdles, though other muscle groups may be involved. Approximately 50% of affected patients show proximal muscle weakness. It is not associated with nutritional, vitamin or mineral deficiencies. The atrophy of the muscle fibres does not lead to an increase in serum creatine kinase and is reversible with 6-12 months abstinence.

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