Encephalopathy due to folate deficiency (Q85626): Difference between revisions

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Os efeitos neuropsiquiátricos reportados da deficiência de folato são marcadamente semelhantes aqueles descritos para deficiência de vitamina B12, embora o metabolismo da vitamina B12 e ácido fólico sejam diferentes. Os pacientes com complicações neuropsiquiátricas por deficiência de folato apresentam-se sem anemia ou macrocitose em relação aos com deficiência de vitamina B12. A causa da deficiência de folato tem sido atribuída de várias maneiras à dieta pobre, doença crônica, uso de drogas (por exemplo, barbitúricos, álcool), má absorção, aumento da demanda, síndromes genéticas raras e específicas ou a causa desconhecida.
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The reported neuropsychiatric effects of folate deficiency are remarkably similar to those described for vitamin-B12 deficiency, though the metabolism of vitamin-B12 and folic-acid differ. More patients with neuropsychiatric complications of folate deficiency present without anaemia or macrocytosis than occurs with vitamin-B12 deficiency. The cause of folate deficiency has been variously ascribed to poor diet, chronic illness, drug use (e.g., barbiturates, alcohol), malabsorption, increased demand, rare and specific genetic syndromes or the cause is unknown.

Revision as of 10:59, 15 August 2026

The reported neuropsychiatric effects of folate deficiency are remarkably similar to those described for vitamin-B12 deficiency, though the metabolism of vitamin-B12 and folic-acid differ. More patients with neuropsychiatric complications of folate deficiency present without anaemia or macrocytosis than occurs with vitamin-B12 deficiency. The cause of folate deficiency has been variously ascribed to poor diet, chronic illness, drug use (e.g., barbiturates, alcohol), malabsorption, increased demand, rare and specific genetic syndromes or the cause is unknown.
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    Encephalopathy due to folate deficiency
    The reported neuropsychiatric effects of folate deficiency are remarkably similar to those described for vitamin-B12 deficiency, though the metabolism of vitamin-B12 and folic-acid differ. More patients with neuropsychiatric complications of folate deficiency present without anaemia or macrocytosis than occurs with vitamin-B12 deficiency. The cause of folate deficiency has been variously ascribed to poor diet, chronic illness, drug use (e.g., barbiturates, alcohol), malabsorption, increased demand, rare and specific genetic syndromes or the cause is unknown.

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