Neonatal peritonitis (Q45920): Difference between revisions
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A peritonite neonatal pode ser de origem bacteriana ou química. A maioria dos casos de peritonite bacteriana é decorrente de perfurações intestinais, ruptura de onfalocele ou necrose intestinal isquêmica. Embora a maioria dos bebês tenha peritonite secundária à perfuração intestinal subsequente à obstrução intestinal, muitos casos são de perfuração sem causa conhecida, possivelmente secundária a defeitos na musculatura intestinal ou isquemia visceral. A peritonite química é menos comum e é devida à perfuração intestinal pré-natal com extrusão de mecônio estéril para a cavidade peritoneal. Os dois tipos podem coexistir se uma perfuração pré-natal permanecer aberta após o nascimento, permitindo a contaminação bacteriana do peritônio previamente estéril. | |||
| description / en | description / en | ||
Neonatal peritonitis may be bacterial or chemical in origin. The majority of cases of bacterial peritonitis are due to intestinal perforations, ruptured omphaloceles, or ischemic intestinal necrosis. Although most babies had peritonitis secondary to intestinal perforation subsequent to intestinal obstruction, many instances are unexplained perforation, possibly secondary to defects in the intestinal musculature or visceral ischemia. The less common chemical peritonitis is due to prenatal intestinal perforation with extrusion of sterile meconium into the peritoneal cavity. The two types may coexist if an antenatal perforation remains open after birth, allowing bacterial contamination of the previously sterile peritoneum. | |||
Revision as of 14:11, 13 August 2026
Neonatal peritonitis may be bacterial or chemical in origin. The majority of cases of bacterial peritonitis are due to intestinal perforations, ruptured omphaloceles, or ischemic intestinal necrosis. Although most babies had peritonitis secondary to intestinal perforation subsequent to intestinal obstruction, many instances are unexplained perforation, possibly secondary to defects in the intestinal musculature or visceral ischemia. The less common chemical peritonitis is due to prenatal intestinal perforation with extrusion of sterile meconium into the peritoneal cavity. The two types may coexist if an antenatal perforation remains open after birth, allowing bacterial contamination of the previously sterile peritoneum.
| Language | Label | Description | Also known as |
|---|---|---|---|
| default for all languages | KB8B |
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| English | Neonatal peritonitis |
Neonatal peritonitis may be bacterial or chemical in origin. The majority of cases of bacterial peritonitis are due to intestinal perforations, ruptured omphaloceles, or ischemic intestinal necrosis. Although most babies had peritonitis secondary to intestinal perforation subsequent to intestinal obstruction, many instances are unexplained perforation, possibly secondary to defects in the intestinal musculature or visceral ischemia. The less common chemical peritonitis is due to prenatal intestinal perforation with extrusion of sterile meconium into the peritoneal cavity. The two types may coexist if an antenatal perforation remains open after birth, allowing bacterial contamination of the previously sterile peritoneum. |
