Vitamin B12 deficiency (Q39923): Difference between revisions

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O vegetarianismo e o quase vegetarianismo imposto pela pobreza são as causas mais comuns de insuficiência nutricional de cobalamina em todo o mundo e em todas as faixas etárias. Em tais populações, o baixo status de cobalamina materna está associado a resultados adversos na gravidez (parto prematuro, retardo de crescimento intrauterino, aborto espontâneo precoce), defeitos do tubo neural, desempenho neurocognitivo reduzido em crianças, remodelação óssea acelerada e baixa densidade mineral óssea com fraturas. A ingestão insuficiente de cobalamina também é observada em bebês amamentados por mães com anemia perniciosa.
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Vegetarianism and poverty-imposed near-vegetarianism are the most common causes of nutritional cobalamin insufficiency worldwide in all age groups. In such populations, low maternal cobalamin status is associated with adverse pregnancy outcomes (preterm birth, intrauterine growth retardation, early recurrent miscarriage), neural tube defects, reduced neurocognitive performance in children, accelerated bone turnover, and low bone mineral density with fractures. Insufficient cobalamin intake is also seen in breast-fed infants of mothers with pernicious anaemia.

Revision as of 05:31, 13 August 2026

Vegetarianism and poverty-imposed near-vegetarianism are the most common causes of nutritional cobalamin insufficiency worldwide in all age groups. In such populations, low maternal cobalamin status is associated with adverse pregnancy outcomes (preterm birth, intrauterine growth retardation, early recurrent miscarriage), neural tube defects, reduced neurocognitive performance in children, accelerated bone turnover, and low bone mineral density with fractures. Insufficient cobalamin intake is also seen in breast-fed infants of mothers with pernicious anaemia.
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    Vitamin B12 deficiency
    Vegetarianism and poverty-imposed near-vegetarianism are the most common causes of nutritional cobalamin insufficiency worldwide in all age groups. In such populations, low maternal cobalamin status is associated with adverse pregnancy outcomes (preterm birth, intrauterine growth retardation, early recurrent miscarriage), neural tube defects, reduced neurocognitive performance in children, accelerated bone turnover, and low bone mineral density with fractures. Insufficient cobalamin intake is also seen in breast-fed infants of mothers with pernicious anaemia.

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