Haemolytic anaemia due to glucose-6-phosphate dehydrogenase deficiency (Q39506): Difference between revisions

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Deficiência da glicose-6-fosfato desidrogenase (G6PD) é a mais comum deficiência hereditária de enzima eritrocitária. Ela pode se manifestar com icterícia neonatal grave que pode levar a sérias consequências neurológicas, ou, mais frequentemente, com anemia hemolítica aguda após ingestão de certos alimentos (feijão de fava), drogas comuns (antimaláricos, sulfamidas, analgésicos) ou durante uma infecção, em indivíduos assintomáticos.
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Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common hereditary erythrocyte enzyme deficiency that can manifest with severe neonatal jaundice which can lead to serious neurological consequences, or, most often, with acute haemolytic anaemia following ingestion of certain foods (fava beans), common drugs (some antimalaria drugs, sulphamides, analgesics), or in the course of an infection, in otherwise asymptomatic individuals.

Revision as of 04:56, 13 August 2026

Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common hereditary erythrocyte enzyme deficiency that can manifest with severe neonatal jaundice which can lead to serious neurological consequences, or, most often, with acute haemolytic anaemia following ingestion of certain foods (fava beans), common drugs (some antimalaria drugs, sulphamides, analgesics), or in the course of an infection, in otherwise asymptomatic individuals.
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    Haemolytic anaemia due to glucose-6-phosphate dehydrogenase deficiency
    Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common hereditary erythrocyte enzyme deficiency that can manifest with severe neonatal jaundice which can lead to serious neurological consequences, or, most often, with acute haemolytic anaemia following ingestion of certain foods (fava beans), common drugs (some antimalaria drugs, sulphamides, analgesics), or in the course of an infection, in otherwise asymptomatic individuals.

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