Glomerular disease with membranous glomerulonephritis (Q101070): Difference between revisions

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Doença glomerular na qual a microscopia de luz mostra espessamento da membrana de filtração que estudos mais aprofundados mostram que é agravada por deposição de imunoglobulina de forma granular fora da membrana basal glomerular (depósitos subepiteliais), que o material da membrana basal possa envolver._x000D_ _x000D_ Tem muitas causas frequentemente associadas à exposição crônica a antígenos ou anticorpos e, na forma idiopática, está frequentemente associada a anticorpos circulantes para o receptor da Fosfolipase A2 (PLA2R).
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Glomerular disease in which light microscopy shows thickening of the filtration membrane which further studies show is contributed to by the deposition of immunoglobulin in a granular fashion outside the glomerular basement membrane (sub-epithelial deposits) which basement membrane material may surround. Has many causes often associated with chronic antigen or antibody exposure, and in the idiopathic form is often associated with circulating antibodies to Phospholipase A2 receptor (PLA2R).

Revision as of 17:51, 16 August 2026

Glomerular disease in which light microscopy shows thickening of the filtration membrane which further studies show is contributed to by the deposition of immunoglobulin in a granular fashion outside the glomerular basement membrane (sub-epithelial deposits) which basement membrane material may surround. Has many causes often associated with chronic antigen or antibody exposure, and in the idiopathic form is often associated with circulating antibodies to Phospholipase A2 receptor (PLA2R).
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ID_817733606
    English
    Glomerular disease with membranous glomerulonephritis
    Glomerular disease in which light microscopy shows thickening of the filtration membrane which further studies show is contributed to by the deposition of immunoglobulin in a granular fashion outside the glomerular basement membrane (sub-epithelial deposits) which basement membrane material may surround. Has many causes often associated with chronic antigen or antibody exposure, and in the idiopathic form is often associated with circulating antibodies to Phospholipase A2 receptor (PLA2R).

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