Cirrhotic cardiomyopathy (Q67638): Difference between revisions

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Cardiomiopatia cirrótica é definida como disfunção cardíaca crônica em pacientes com cirrose, caracterizada por resposta contrátil embotada ao estresse/exercício e/ou relaxamento diastólico alterado com anormalidades eletrofisiológicas, na ausência de doença cardíaca conhecida e independentemente da causa da cirrose, embora algumas etiologias (p.ex., sobrecarga de ferro e consumo de álcool) impactem adicionalmente na estrutura e função do miocárdio. É considerada relacionada a hipertensão portal e cirrose com alterações intrínsecas da função miocárdica.
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Cirrhotic cardiomyopathy is defined as chronic cardiac dysfunction in patients with cirrhosis characterised by blunted contractile responsiveness to stress/exercise and-or altered diastolic relaxation with electrophysiological abnormalities, in the absence of known cardiac disease and irrespective of the cause of cirrhosis, although some aetiologies (e.g., iron overload and alcohol consumption) further impact on myocardial structure and function. It is considered to be related to both portal hypertension and cirrhosis with intrinsic alterations in myocardial function.

Revision as of 15:28, 14 August 2026

Cirrhotic cardiomyopathy is defined as chronic cardiac dysfunction in patients with cirrhosis characterised by blunted contractile responsiveness to stress/exercise and-or altered diastolic relaxation with electrophysiological abnormalities, in the absence of known cardiac disease and irrespective of the cause of cirrhosis, although some aetiologies (e.g., iron overload and alcohol consumption) further impact on myocardial structure and function. It is considered to be related to both portal hypertension and cirrhosis with intrinsic alterations in myocardial function.
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ID_1268082489
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    Cirrhotic cardiomyopathy
    Cirrhotic cardiomyopathy is defined as chronic cardiac dysfunction in patients with cirrhosis characterised by blunted contractile responsiveness to stress/exercise and-or altered diastolic relaxation with electrophysiological abnormalities, in the absence of known cardiac disease and irrespective of the cause of cirrhosis, although some aetiologies (e.g., iron overload and alcohol consumption) further impact on myocardial structure and function. It is considered to be related to both portal hypertension and cirrhosis with intrinsic alterations in myocardial function.

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