Oedema due to increased capillary pressure (Q47532): Difference between revisions
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O aumento da pressão capilar aumenta o vazamento de fluido do compartimento vascular para os tecidos intersticiais, resultando em edema. Causas incluem o comprometimento ou obstrução do retorno venoso (sobrecarga de fluido, trombose venosa, insuficiência cardíaca direita, compressão venosa por tumor), aumento do fluxo sanguíneo (resposta fisiológica à exposição ao calor, malformações arteriovenosas, controle vasomotor cutâneo prejudicado devido a drogas ou neuropatia autonômica), ou pressão oncótica plasmática reduzida por hipoproteinemia. | |||
| description / en | description / en | ||
Increased capillary pressure increases the leakage of fluid from the vascular compartment to the interstitial tissues, resulting in oedema. Causes include impaired or obstructed venous return (fluid overload, venous thrombosis, right heart failure, venous compression from tumour tissue), increased blood flow (physiological response to heat exposure, arteriovenous malformations, disturbed cutaneous vasomotor control due to drug or autonomic neuropathy), or reduced plasma oncotic pressure due to hypoproteinaemia. | |||
Revision as of 16:45, 13 August 2026
Increased capillary pressure increases the leakage of fluid from the vascular compartment to the interstitial tissues, resulting in oedema. Causes include impaired or obstructed venous return (fluid overload, venous thrombosis, right heart failure, venous compression from tumour tissue), increased blood flow (physiological response to heat exposure, arteriovenous malformations, disturbed cutaneous vasomotor control due to drug or autonomic neuropathy), or reduced plasma oncotic pressure due to hypoproteinaemia.
| Language | Label | Description | Also known as |
|---|---|---|---|
| default for all languages | MG29.10 |
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| English | Oedema due to increased capillary pressure |
Increased capillary pressure increases the leakage of fluid from the vascular compartment to the interstitial tissues, resulting in oedema. Causes include impaired or obstructed venous return (fluid overload, venous thrombosis, right heart failure, venous compression from tumour tissue), increased blood flow (physiological response to heat exposure, arteriovenous malformations, disturbed cutaneous vasomotor control due to drug or autonomic neuropathy), or reduced plasma oncotic pressure due to hypoproteinaemia. |
