Duodenitis (Q43267): Difference between revisions

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Duodenite é uma lesão da mucosa duodenal que envolve dano epitelial e inflamação da mucosa, exceto por qualquer defeito epitelial. A duodenite é causada por vários fatores como elevada secreção ácida, agentes infecciosos, drogas, agentes químicos e outros. A metaplasia gástrica é considerada resposta adaptativa à hiperacidez. Helicobacter pylori pode colonizar o epitélio da metaplasia gástrica, e induzir a duodenite. A duodenite é diagnosticada por histopatologia e/ou endoscopia. A duodenite é classificada como fase aguda e crônica conforme o curso clínico.
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Duodenitis is an injury of duodenal mucosa that involves epithelial damage and mucosal inflammation except for any epithelial defect. Duodenitis is caused by various factors such as high acid secretion, infectious agents, drugs, chemical agents and others. Gastric metaplasia (GM) is considered adaptive responses to hyperacidity. Helicobacter pylori can be colonized on GM epithelium, and induce duodenitis. Duodenitis is diagnosed histopathologically and/or endoscopically. Duodenitis is classified as acute and chronic phase by clinical course.

Revision as of 10:12, 13 August 2026

Duodenitis is an injury of duodenal mucosa that involves epithelial damage and mucosal inflammation except for any epithelial defect. Duodenitis is caused by various factors such as high acid secretion, infectious agents, drugs, chemical agents and others. Gastric metaplasia (GM) is considered adaptive responses to hyperacidity. Helicobacter pylori can be colonized on GM epithelium, and induce duodenitis. Duodenitis is diagnosed histopathologically and/or endoscopically. Duodenitis is classified as acute and chronic phase by clinical course.
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    Duodenitis
    Duodenitis is an injury of duodenal mucosa that involves epithelial damage and mucosal inflammation except for any epithelial defect. Duodenitis is caused by various factors such as high acid secretion, infectious agents, drugs, chemical agents and others. Gastric metaplasia (GM) is considered adaptive responses to hyperacidity. Helicobacter pylori can be colonized on GM epithelium, and induce duodenitis. Duodenitis is diagnosed histopathologically and/or endoscopically. Duodenitis is classified as acute and chronic phase by clinical course.

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