Hypervitaminosis D (Q39979): Difference between revisions
From determinar.ia.br - Determine suas informações
Created a new Item |
Changed label, description and/or aliases in pt-br, en |
||
| description / pt-br | description / pt-br | ||
A hipervitaminose D é secundária à ingestão excessiva de vitamina D. Pode ocorrer com ingestão elevada a longo prazo ou por ingestão aguda em excesso. O excesso de quantidades resulta em concentrações anormalmente altas de cálcio e fosfato no soro. Os sinais e sintomas da intoxicação por vitamina D são secundários à hipercalcemia. As manifestações gastrointestinais incluem náuseas, vômitos, constipação, dor abdominal e pancreatite. Os possíveis achados cardíacos são hipertensão, diminuição do intervalo Q-T e arritmias. Os efeitos da hipercalcemia no sistema nervoso central incluem letargia, hipotonia, confusão, desorientação, depressão, psicose, alucinações e coma. A hipercalcemia danifica os mecanismos de concentração renal, o que pode levar à poliúria, desidratação e hipernatremia. A hipercalcemia também pode causar insuficiência renal aguda, nefrolitíase e nefrocalcinose, que pode resultar em insuficiência renal crônica. As mortes geralmente estão associadas a arritmias ou desidratação. | |||
| description / en | description / en | ||
Hypervitaminosis D is secondary to excessive intake of vitamin D. It can occur with long-term high intake or with a substantial, acute ingestion. Excess amounts result in abnormally high concentrations of calcium and phosphate in the serum. The signs and symptoms of vitamin D intoxication are secondary to hypercalcaemia. Gastrointestinal manifestations include nausea, vomiting, constipation, abdominal pain and pancreatitis. Possible cardiac findings are hypertension, decreased Q-T interval and arrhythmias. The central nervous system effects of hypercalcaemia include lethargy, hypotonia, confusion, disorientation, depression, psychosis, hallucinations and coma. Hypercalcaemia impairs renal concentrating mechanisms, which can lead to polyuria, dehydration and hypernatremia. Hypercalcaemia can also lead to acute renal failure, nephrolithiasis and nephrocalcinosis, which can result in chronic renal insufficiency. Deaths are usually associated with arrhythmias or dehydration. | |||
Revision as of 05:36, 13 August 2026
Hypervitaminosis D is secondary to excessive intake of vitamin D. It can occur with long-term high intake or with a substantial, acute ingestion. Excess amounts result in abnormally high concentrations of calcium and phosphate in the serum. The signs and symptoms of vitamin D intoxication are secondary to hypercalcaemia. Gastrointestinal manifestations include nausea, vomiting, constipation, abdominal pain and pancreatitis. Possible cardiac findings are hypertension, decreased Q-T interval and arrhythmias. The central nervous system effects of hypercalcaemia include lethargy, hypotonia, confusion, disorientation, depression, psychosis, hallucinations and coma. Hypercalcaemia impairs renal concentrating mechanisms, which can lead to polyuria, dehydration and hypernatremia. Hypercalcaemia can also lead to acute renal failure, nephrolithiasis and nephrocalcinosis, which can result in chronic renal insufficiency. Deaths are usually associated with arrhythmias or dehydration.
| Language | Label | Description | Also known as |
|---|---|---|---|
| default for all languages | 5B90.2 |
||
| English | Hypervitaminosis D |
Hypervitaminosis D is secondary to excessive intake of vitamin D. It can occur with long-term high intake or with a substantial, acute ingestion. Excess amounts result in abnormally high concentrations of calcium and phosphate in the serum. The signs and symptoms of vitamin D intoxication are secondary to hypercalcaemia. Gastrointestinal manifestations include nausea, vomiting, constipation, abdominal pain and pancreatitis. Possible cardiac findings are hypertension, decreased Q-T interval and arrhythmias. The central nervous system effects of hypercalcaemia include lethargy, hypotonia, confusion, disorientation, depression, psychosis, hallucinations and coma. Hypercalcaemia impairs renal concentrating mechanisms, which can lead to polyuria, dehydration and hypernatremia. Hypercalcaemia can also lead to acute renal failure, nephrolithiasis and nephrocalcinosis, which can result in chronic renal insufficiency. Deaths are usually associated with arrhythmias or dehydration. |
