Hereditary methaemoglobinaemia (Q39674): Difference between revisions

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Metemoglobinemia hereditária é um transtorno raro da hemácia classificada principalmente em dois fenótipos clínicos: metemoglobinemia congênita autossômica recessiva (ou hereditária) tipos I e II (RCM/RHM tipo I; RCM/RHM tipo 2). Na RCM tipo 1, cianose bem tolerada desde o nascimento é o único sintoma. RCM tipo 2, com perda global da função do Cb5R, é muito mais grave; a cianose é acompanhada por disfunção neurológica (com déficit intelectual, microcefalia, retardo do crescimento, opistótono, estrabismo e hipertonia), que geralmente se torna evidente durante os primeiros quatro meses de vida.
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Hereditary methemoglobinemia (HM) is a rare red cell disorder classified principally into two clinical phenotypes: autosomal recessive congenital (or hereditary) methemoglobinemia types I and II (RCM/RHM type 1; RCM/RHM type 2). In RCM type 1, well-tolerated cyanosis from birth is the only symptom. RCM type 2, with global loss of Cb5R function, is much more severe; the cyanosis is accompanied by neurological dysfunction (with intellectual deficit, microcephaly, growth retardation, opisthotonus, strabismus and hypertonia), which usually becomes evident during the first four months of life.

Revision as of 05:10, 13 August 2026

Hereditary methemoglobinemia (HM) is a rare red cell disorder classified principally into two clinical phenotypes: autosomal recessive congenital (or hereditary) methemoglobinemia types I and II (RCM/RHM type 1; RCM/RHM type 2). In RCM type 1, well-tolerated cyanosis from birth is the only symptom. RCM type 2, with global loss of Cb5R function, is much more severe; the cyanosis is accompanied by neurological dysfunction (with intellectual deficit, microcephaly, growth retardation, opisthotonus, strabismus and hypertonia), which usually becomes evident during the first four months of life.
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    Hereditary methaemoglobinaemia
    Hereditary methemoglobinemia (HM) is a rare red cell disorder classified principally into two clinical phenotypes: autosomal recessive congenital (or hereditary) methemoglobinemia types I and II (RCM/RHM type 1; RCM/RHM type 2). In RCM type 1, well-tolerated cyanosis from birth is the only symptom. RCM type 2, with global loss of Cb5R function, is much more severe; the cyanosis is accompanied by neurological dysfunction (with intellectual deficit, microcephaly, growth retardation, opisthotonus, strabismus and hypertonia), which usually becomes evident during the first four months of life.

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