Certain specified syndrome of delayed hypoxic cerebral demyelination (Q86970): Difference between revisions

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+Leucoencefalopatia pós-hipóxica tardia é uma síndrome desmielinizante caracterizada por início agudo de sintomas neuropsiquiátricos dias a semanas após recuperação aparente de coma após um período de hipo-oxigenação cerebral prolongada. Deterioração neurológica aguda e desmielinização cerebral que ocorrem dias a semanas após a recuperação clínica de um insulto hipóxico-isquêmico cerebral. Achados na RNM demonstram hiperintensidade difusa da substância branca cerebral em sequências T2, particularmente na região dorsal dos lobos frontal e parietal.
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+Delayed post-hypoxic leukoencephalopathy is a demyelinating syndrome characterised by acute onset of neuropsychiatric symptoms days to weeks following apparent recovery from coma after a period of prolonged cerebral hypo-oxygenation. Acute neurologic deterioration and cerebral demyelination that occur days to weeks after the clinical recovery from a hypoxic-ischemic insult in the brain . MRI findings demonstrate diffuse hyperintensity of the cerebral white matter on T2-sequences, particularly in the region of the dorsal frontal and parietal lobes.
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Latest revision as of 12:32, 15 August 2026

Delayed post-hypoxic leukoencephalopathy is a demyelinating syndrome characterised by acute onset of neuropsychiatric symptoms days to weeks following apparent recovery from coma after a period of prolonged cerebral hypo-oxygenation. Acute neurologic deterioration and cerebral demyelination that occur days to weeks after the clinical recovery from a hypoxic-ischemic insult in the brain . MRI findings demonstrate diffuse hyperintensity of the cerebral white matter on T2-sequences, particularly in the region of the dorsal frontal and parietal lobes.

Language	Label	Description	Also known as
default for all languages	ID_1641401568
English	Certain specified syndrome of delayed hypoxic cerebral demyelination	Delayed post-hypoxic leukoencephalopathy is a demyelinating syndrome characterised by acute onset of neuropsychiatric symptoms days to weeks following apparent recovery from coma after a period of prolonged cerebral hypo-oxygenation. Acute neurologic deterioration and cerebral demyelination that occur days to weeks after the clinical recovery from a hypoxic-ischemic insult in the brain . MRI findings demonstrate diffuse hyperintensity of the cerebral white matter on T2-sequences, particularly in the region of the dorsal frontal and parietal lobes.