Myelopathy due to excess of zinc (Q89797): Difference between revisions

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+August 2026Timestamp +2026-08-13T00:00:00Z
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Latest revision as of 16:38, 15 August 2026

Zinc-induced myeloneuropathy was recently (re)discovered and its pathophysiology elaborated as resulting from secondary copper deficiency. Excess zinc blocks copper absorption by binding to copper molecules through intestinal metallothionein. Oral intake of zinc in excess of the minimum daily requirement (approximately 15 mg/d) can deplete total body copper stores, but this treatment may take years to show effect.

Language	Label	Description	Also known as
default for all languages	ID_1935468582
English	Myelopathy due to excess of zinc	Zinc-induced myeloneuropathy was recently (re)discovered and its pathophysiology elaborated as resulting from secondary copper deficiency. Excess zinc blocks copper absorption by binding to copper molecules through intestinal metallothionein. Oral intake of zinc in excess of the minimum daily requirement (approximately 15 mg/d) can deplete total body copper stores, but this treatment may take years to show effect.