Idiopathic central alveolar hypoventilation (Q41271): Difference between revisions
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Hipoventilação alveolar central idiopática é definida como a presença de ventilação alveolar reduzida resultando em hipercapnia e hipoxemia relacionadas ao sono em indivíduos com propriedades mecânicas do pulmão e da bomba respiratória normais presumidas. Hipoventilação crônica durante o sono existe sem qualquer comprometimento prontamente identificável da respiração, tal como condições de vias aéreas ou parênquima pulmonares, anormalidades neurológicas, neuromusculares ou de parede torácica, obesidade grave, outro transtorno respiratório relacionado ao sono, ou uso de medicamentos ou substâncias depressivos respiratórios. Tanto a hipoventilação diurna como a noturna são consideradas como primariamente devidas a quimiorresponsividade embotada ao dióxido de carbono (CO2) e ao oxigênio (O2). Os pacientes podem se queixar de cefaleia matinal, fadiga, declínio neurocognitivo e distúrbio do sono, ou podem ser completamente assintomáticos._x000D_ _x000D_ Nota: Um diagnóstico definitivo requer a evidência objetiva baseada em polissonografia com monitorização do dióxido de carbono (CO2) por medidas arterial, tidal final ou transcutânea. | |||||||||||||||
| description / en | description / en | ||||||||||||||
Idiopathic central alveolar hypoventilation is defined as the presence of decreased alveolar ventilation resulting in sleep related hypercapnia and hypoxemia in individuals with presumed normal mechanical properties of the lung and respiratory pump. Chronic hypoventilation during sleep exists without any readily identifiable impairments of respiration, such as pulmonary airway or parenchymal conditions, neurologic, neuromuscular or chest wall abnormalities, severe obesity, other sleep related breathing disorder, or use of respiratory depressant medications or substances. Diurnal as well as nocturnal hypoventilation is believed to be due primarily to blunted chemoresponsiveness to carbon dioxide (CO2) and oxygen (O2). Patients may complain of morning headaches, fatigue, neurocognitive decline and sleep disturbance, or may be entirely asymptomatic. Note: A definitive diagnosis requires objective evidence based on polysomnography with carbon dioxide (CO2) monitoring (by arterial, end-tidal or transcutaneous measures). | |||||||||||||||
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| Property / Canonical URI: https://id.who.int/icd/entity/1024837047 / rank | |||||||||||||||
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CID11:7A42.3 | |||||||||||||||
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dki-india-7A42.3 | |||||||||||||||
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| Property / Knowledge Architect: https://pauloleads.com.br/cases-publicos/ / rank | |||||||||||||||
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13 August 2026
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| Property / Collection date: 13 August 2026 / rank | |||||||||||||||
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| Property / Linked ICD 10: G47.3 / rank | |||||||||||||||
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Latest revision as of 07:22, 13 August 2026
Idiopathic central alveolar hypoventilation is defined as the presence of decreased alveolar ventilation resulting in sleep related hypercapnia and hypoxemia in individuals with presumed normal mechanical properties of the lung and respiratory pump. Chronic hypoventilation during sleep exists without any readily identifiable impairments of respiration, such as pulmonary airway or parenchymal conditions, neurologic, neuromuscular or chest wall abnormalities, severe obesity, other sleep related breathing disorder, or use of respiratory depressant medications or substances. Diurnal as well as nocturnal hypoventilation is believed to be due primarily to blunted chemoresponsiveness to carbon dioxide (CO2) and oxygen (O2). Patients may complain of morning headaches, fatigue, neurocognitive decline and sleep disturbance, or may be entirely asymptomatic. Note: A definitive diagnosis requires objective evidence based on polysomnography with carbon dioxide (CO2) monitoring (by arterial, end-tidal or transcutaneous measures).
| Language | Label | Description | Also known as |
|---|---|---|---|
| default for all languages | 7A42.3 |
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| English | Idiopathic central alveolar hypoventilation |
Idiopathic central alveolar hypoventilation is defined as the presence of decreased alveolar ventilation resulting in sleep related hypercapnia and hypoxemia in individuals with presumed normal mechanical properties of the lung and respiratory pump. Chronic hypoventilation during sleep exists without any readily identifiable impairments of respiration, such as pulmonary airway or parenchymal conditions, neurologic, neuromuscular or chest wall abnormalities, severe obesity, other sleep related breathing disorder, or use of respiratory depressant medications or substances. Diurnal as well as nocturnal hypoventilation is believed to be due primarily to blunted chemoresponsiveness to carbon dioxide (CO2) and oxygen (O2). Patients may complain of morning headaches, fatigue, neurocognitive decline and sleep disturbance, or may be entirely asymptomatic. Note: A definitive diagnosis requires objective evidence based on polysomnography with carbon dioxide (CO2) monitoring (by arterial, end-tidal or transcutaneous measures). |
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CID11:7A42.3
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dki-india-7A42.3
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Concluído
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13 August 2026
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